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. rarQ:

On Amazon S3 Getting Error “Access Denied” when a call to IsObjectAllowed is made from a Service Workers Registration

I have the following manifest.json:
{
“asset-manifest-inline”: true,
“exclude-ext”: [“html”],
“manifest-version”: 2,
“name”: “test”
}

I’m registering Service Workers with:
sw.js
sw.js
I’m authenticating calls to /sw.js and /assets/test.js with:
sw.js
sw.js
Then I’m calling this:
sw.js
What am I doing wrong?

A:

Based on the logs, it looks like you are requiring your assets from the root of your site. For instance:
@import ‘/assets/test.js’;

If you can, consider setting your manifest’s assets-base to the root of your site. This will ensure your assets are being loaded from the correct path.
“assets-base”: “/assets”

Documentation is here

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References

Category:1937 births
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The quantitative analyses of the lichen thalli from “chlorococcus” (Hypogymnia physodes (L.) Link. and H. fluviatilis (L.) Hoffm. & Link.) obtained at different dates and salinities have been carried out. Only the mircrotubules of the cortex of H. physodes have been revealed, whereas the detailed structure of the cortex of H. fluviatilis has not been characterized before. The data obtained are used to discuss the possibility of the biogeocenosis of lichens.Inhibition of Angiotensin-converting Enzyme and Its Therapeutic Use.
Currently, the only FDA-approved drugs for the management of heart failure (HF) are B-type natriuretic peptide and angiotensin-converting enzyme (ACE) inhibitors. Increasing evidence has demonstrated that medications targeting the renin-angiotensin-aldosterone system could promote ventricular remodeling and fibrosis, with considerable risks. ACE2 is a homolog of ACE, with a completely opposite effect to ACE. Mounting evidence has shown that ACE2/Ang (1-7)/Mas signaling pathway is involved in the pathological process of cardiovascular diseases and may be a promising therapeutic target for HF. In this review, we summarize the changes and role of ACE and ACE2 in the development of HF. Sub
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